skip to content »

Pathology

Houston, Texas

Department of Pathology
Pathology
not shown on screen

Faculty

G. Jackson Snipes, M.D., Ph.D.Associate Professor

photoAlt

Education

  • M.D., Vanderbilt University School of Medicine, 1988
  • Ph.D., Vanderbilt University, 1986

Training

Internship

  • Pathology, Stanford University, 1989

Residency

  • Pathology, Stanford University, 1990

Fellowship

  • Neuropathology, Stanford University, 1991-1992

Post-doctoral Training

  • Neurobiology, Stanford University, 1992-1995

Specialty

  • Neuropathology

Board Certification

  • Anatomic Pathology
  • Neuropathology

Area of Interest

  • General Neuropathology
  • Neuromuscular Disorders

Current Position

  • Associate Professor of Pathology, Baylor College of Medicine
  • Adjunct Associate Professor of Molecular Physiology and Biophysics

Professional Positions Held

  • Adjunct Associate Professor of Molecular Physiology and Biophysics, Baylor College of Medicine, 2006-present
  • Associate Professor of Pathology, Baylor College of Medicine, 2001-present
  • Associate Professor of Pathology, Neurology and Neurosurgery, and Cell Biology-McGill University, 2001
  • Assistant Professor of Pathology, Neurology and Neurosurgery, and Cell Biology-McGill University, 1995-2001
  • Assistant Professor of Pathology, Acting, Stanford University, 1992-1995

Other Positions

Honors and Awards

Membership on professional organizations

  • American Association of Neuropathologists
  • American Academy of Neurology
  • Society for Neuroscience
  • College of American Pathologists

Representative Publications

  1. Comati, A., Beck, H., Halliday, W.., Snipes, G. J., Plate, K. H., and T. Acker. Upregulation of hypoxia inducible factor (HIF)-1a in leptomeningeal vascular malformations of Sturge-Weber Syndrome. J. Neuropathol. Exp. Neurol. in press.
  2. Khajavi, M., Inoue, K., Wiszniewski, W., Ohyama, T., Snipes, G. J., and J. R. Lupski. (2005) Curcumin Treatment Abrogates Endoplasmic Reticulum Retention and Aggregation-Induced Apoptosis Associated with Neuropathy-Causing Myelin Protein Zero-Truncating Mutants. Am. J. Hum. Genet. 77:841-850.
  3. Orfali, W., Nicholson, R. N., Guiot, M.-C., Peterson, A. C. and G. J. Snipes. (2005) 8.5 kb of the PMP22 Promoter Targets Transgene Expression to the Peripheral Nervous System in transgenic mice. J. Neurosci. Res. 80:37-46.
  4. D'Agostino, M.D., Bastos, A., Piras, C., Bernasconi, A., Grisar, T., Zur, V.G., Snipes, J., Juhasz, C., Chugani, H., Guerrini, H., Cross, H., Andermann, E., Dubeau, F., Montes, J., Olivier, Andermann, R. (2004) Posterior quadrantic dysplasia or hemi-hemimegalencephaly: A characteristic brain malformation. Neurology, 62:2214-2220.
  5. Guerreiro, M. M., Quesney, L.F., Salanova, V. and G. J. Snipes. (2003) Continuous Electrocorticogram Epileptiform Discharges due to Brain Gliosis. J. Clin. Neurophys. 20:239-242.
  6. Shames, I., Fraser, A., Colby, J., Orfali, W., and G. J. Snipes. (2003) Phenotypic differences between Peripheral Myelin Protein-22 (PMP-22) and Protein zero (P0) mutations associated with Charcot-Marie-Tooth and related diseases. J. Neuropathol. Exp. Neurol. 62: 751-764.
  7. Senger, D. L., Tudan, C., Guiot, M.-C., Mazzoni, I. E., Molenkamp , G., LeBlanc, R., Antel, J., Olivier, A. Snipes, G.J. and Kaplan, D. R. (2002) Suppression of Rac activity induces apoptosis of human glioma cells but not normal human astrocytes. Cancer Res, 62:2131-2140.
  8. Dickson, K., Bergeron, J.J.M., Shames, I, Colby, J., Nguyen, D.T., Chevet, E., Thomas, D.Y., and G. J. Snipes. (2002) Association of calnexin with mutant peripheral myelin protein-22 (PMP-22) ex vivo: A basis for “gain of function” ER diseases. Proc. Natl. Acad. Sci (USA) 99:9852-9857.
  9. Colby, J., Nicholson, R., Dickson, K. M., Orfali, W., Naef, R., Suter, U., and G. J. Snipes. (2000) PMP22 carrying the Trembler or Trembler-J mutation is intracellularly retained in myelinating Schwann cells. Neurobiol. of Disease,7:561-573.